So far in this series we've focused on a new way of understanding diabesity as an autoimmune, inflammatory disorder, and we've looked in a general way at the underlying mechanisms (inflammation, genetics, environmental triggers and leaky gut) that contribute to diabesity.
Now that we've laid that foundation, we're going to take a closer look at some of those mechanisms. In this article, we'll discuss the three major dietary toxins that trigger diabesity:
Cereal grains (especially refined flour)
Omega-6 ...
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In the previous article in this series, I argued that diabesity is an autoimmune, inflammatory disorder. In this article, we're going to review the evidence linking inflammation to obesity and type 2 diabetes (T2DM) and learn why inflammation may be the single-most important mechanism driving the diabesity epidemic.
The inflammation-diabesity connection is a hot topic in the scientific literature. A Pubmed search for "inflammation diabetes obesity" turns up more than 1,800 articles. The association between ...
In this article, I'd like to present a unified model of diabesity. I think it's helpful to have a "birds-eye view" of how diabesity begins and progresses. It gives us a context for the articles that will follow, each of which will examine specific elements of the model in much more detail.
Two caveats before we dive in. First, remember that all models are imperfect. They're useful tools, but as the saying ...
Obesity, insulin resistance, metabolic syndrome and type 2 diabetes have reached epidemic proportions. There's not a person reading this article who isn't affected by these conditions, either directly or indirectly. Yet as common as these conditions are, few people understand how closely they're related to one another.
It is now clear that not only do these conditions share the same underlying causes - and thus require the same treatment - they are 100% preventable and, ...
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